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• F-B l, M J, F-P M, A i, r-F J, P c
Most relevant ernándeZertolínulloluenteSrAdoloBidocAerrericAdoet
Al.. Deficient glucocorticoid induction of anti-inflammatory genes in nasal polyp fi-
scientific broblasts of asthmatic patients with and without aspirin intolerance. J Allergy Clin

articles
Immunol. 2013 Nov;132(5):1243-1246.e12.

• torreS r, HerreriAS A, SerrA-PAgèS M, MArco A, PlAZA J, coStA-FArré c et Al.. Locally

administered prostaglandin E2 prevents aeroallergen-induced airway sensitization in
mice through immunomodulatory mechanisms. Pharmacol Res. 2013 Apr;70(1):50-9.

• g M, r d, M-M M, r-F J, P l, P c .. Lung myo-
ABASAoYoolinAolinAocAerreruJolSicAdoet Al
fibroblasts are characterized by down-regulated cyclooxygenase-2 and its main me-
tabolite, prostaglandin E2. PLoS One. 2013;8(6):e65445.

• l t, u M, Y c Y, Y H, d l, X A .. Telomerase and telo-
iullenBrucHoungHoiuingAuBetet Al
mere length in pulmonary fibrosis. Am J Respir Cell Mol Biol. 2013 Aug;49(2):260-8.

• AlMudéVer P, MilArA J, de diego A, SerrAno-MollAr A, XAuBet A, PéreZ-ViZcAino F. Role of
tetrahydrobiopterin in pulmonary vascular remodelling associated with pulmonary

fibrosis. Thorax. 2013 Oct;68(10):938-48.

Five active competitive research projects (Instituto Carlos III) whose principal
Highlights
investigators are members of the group. Five active clinical assays in asthma
and two in idiopathic lung fibrosis. Twenty four publications in indexed journals,

nine of them published in journals of the first quartile. Members of the group

have participated as authors in three international clinical guides (ARIA/rhinitis
and asthma, EPOS/chronic rhinosinusitis and nasal polyposis, and EACCI-aspirin

intolerant asthma), and in two national guides (Gema/asma; SEPAR/Idiopathic

Lug Fibrosis). The studies of the group have produced two patents: a) trans-
plantation of type II alveolar cells in the treatment of idiopathic lung fibrosis.

2. Synergistic combination of an anti-IgE and EP2 receptor agonist in asthma.

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