Page 161 - MemoriaER-Eng
P. 161




www.ciberer.es


We have performed transcriptomic and proteomic studies (in collaboration with 
Highlights
the group of CIBERER U732) of various cellular models of frataxin deficiency that 

have allowed us to identify some candidates that could serve as biomarkers and 
therapeutic targets for Friedreich’s ataxia.

We have initiated two projects aimed at exploring novel approaches for the the- 

rapy of Friedreich ataxia, one focused on the development of gene therapy and 
another addressing the use of agonists of neurotrophic factor receptors.























































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